Term Information

Accession
GO:0002755
Name
MyD88-dependent toll-like receptor signaling pathway
Ontology
biological_process
Synonyms
MyD88-dependent TLR signaling pathway, MyD88-dependent toll-like receptor signalling pathway
Alternate IDs
None
Definition
A toll-like receptor signaling pathway in which the MyD88 adaptor molecule mediates transduction of the signal. Toll-like receptors directly bind pattern motifs from a variety of microbial sources to initiate an innate immune response. Source: PMID:12467241, PMID:12524386, PMID:15585605, ISBN:0781735149, PMID:15728447, GOC:add, PMID:12855817
Comment
None
History
See term history for GO:0002755 at QuickGO
Subset
None
Feedback
Comments, changes to terms, or requests for new ontology terms can be made at GO issue tracker on GitHub.

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Parents of MyD88-dependent toll-like receptor signaling pathway (GO:0002755)
subject[Reorder by subject] relation[Reorder by relation] object[Reorder by object]
MyD88-dependent toll-like receptor signaling pathway [is_a relation] is_a  toll-like receptor signaling pathway (GO:0002224)
Children of MyD88-dependent toll-like receptor signaling pathway (GO:0002755)
subject[Reorder by subject] relation[Reorder by relation] object[Reorder by object]
regulation of MyD88-dependent toll-like receptor signaling pathway (GO:0034124) [RO:0002211 relation] RO:0002211  MyD88-dependent toll-like receptor signaling pathway
positive regulation of MyD88-dependent toll-like receptor signaling pathway (GO:0034126) [RO:0002213 relation] RO:0002213  MyD88-dependent toll-like receptor signaling pathway
negative regulation of MyD88-dependent toll-like receptor signaling pathway (GO:0034125) [RO:0002212 relation] RO:0002212  MyD88-dependent toll-like receptor signaling pathway
MyD88-dependent toll-like receptor 4 signaling pathway (GO:0035660) [is_a relation] is_a  MyD88-dependent toll-like receptor signaling pathway
MyD88-dependent toll-like receptor 2 signaling pathway (GO:0035661) [is_a relation] is_a  MyD88-dependent toll-like receptor signaling pathway
None.